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Urinary 1-hydroxypyrene in occupationally-exposed and non-exposed individuals in Silesia, Poland

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Urinary 1-hydroxypyrene in occupationally-exposed and non-exposed individuals in Silesia, Poland

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dc.contributor.author Zając, Joanna [SAP20002658] pl
dc.contributor.author Gomółka, Ewa [SAP20008191] pl
dc.contributor.author Szot, Wojciech [SAP20001581] pl
dc.date.accessioned 2020-01-17T09:20:34Z
dc.date.available 2020-01-17T09:20:34Z
dc.date.issued 2018 pl
dc.identifier.issn 1232-1966 pl
dc.identifier.uri https://ruj.uj.edu.pl/xmlui/handle/item/142488
dc.language eng pl
dc.rights Udzielam licencji. Uznanie autorstwa - Użycie niekomercyjne 3.0 Polska *
dc.rights.uri http://creativecommons.org/licenses/by-nc/3.0/pl/legalcode *
dc.title Urinary 1-hydroxypyrene in occupationally-exposed and non-exposed individuals in Silesia, Poland pl
dc.type JournalArticle pl
dc.description.physical 625-629 pl
dc.abstract.en Introduction. The aim of presented study was comparison of urinary 1-hydroxypyrene concentration among coke plant workers (before and after working week) and among non-exposed individuals from the same area, taking smoking status into consideration. Materials and method. 647 coke plant workers and 206 individuals living in the same area were analysed with respect to urinary 1-OHP concentration and smoking status. Urinary samples were measured using high performance liquid chromatography (HPLC) with fluorescent detection. Concentrations were normalized with respect to creatinine concentration. For workers, two samples were collected before and after working week. Multiple regression was performed to distinguish and quantify the influence of cigarette smoking and occupational PAH exposition on the urinary 1-OHP levels. Results. Average urinary 1-OHP concentration of samples collected before the working week was: 1.07 μg/g; after the working week: 2.36 μg/g and for control: 0.74 μg/g. The samples collected at the beginning of the working week were not suitable for assessment of the workers’ background (non-occupational) exposition. Smoking cigarettes induced a rise in urinary 1-OHP level by 16%, on average (CI: 5% – 28%), and working for a whole working week at the coke plant made urinary 1-OHP levels, on average, 3.21 times higher (CI: 2.91 – 3.54). Conclusions. Working at the coke plant increases significantly urinary 1-OHP concentration in comparison to nonoccupationally exposed individuals, both for samples collected before and after the working week. Smoking remains a significant source of PAHs exposition, despite the fact that occupational exposure is greater. Health promotion programmes should address both the occupational health risks reduction and smoking prevention. pl
dc.subject.en 1-hydroxypyrene pl
dc.subject.en pyrene pl
dc.subject.en coke plant pl
dc.subject.en cigarettes smoking pl
dc.description.volume 25 pl
dc.description.number 4 pl
dc.description.points 30 pl
dc.identifier.doi 10.26444/aaem/75940 pl
dc.identifier.eissn 1898-2263 pl
dc.title.journal Annals of Agricultural and Environmental Medicine pl
dc.language.container eng pl
dc.affiliation Wydział Lekarski : Zakład Higieny i Dietetyki pl
dc.affiliation Wydział Lekarski : Katedra Toksykologii i Chorób Środowiskowych pl
dc.subtype Article pl
dc.rights.original CC-BY-NC; otwarte czasopismo; ostateczna wersja wydawcy; w momencie opublikowania; 0 pl
dc.identifier.project ROD UJ / OP pl
dc.cm.id 87499
dc.cm.date 2020-01-07
.pointsMNiSW [2018 A]: 30


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Udzielam licencji. Uznanie autorstwa - Użycie niekomercyjne 3.0 Polska Except where otherwise noted, this item's license is described as Udzielam licencji. Uznanie autorstwa - Użycie niekomercyjne 3.0 Polska